![]() ![]() Voriconazole-induced retinoid-like photosensitivity in children. Photoaging and phototoxicity from long-term voriconazole treatment in a 15-year-old girl. Racette AJ, Roenigk HH Jr, Hansen R, Mendelson D, Park A. Voriconazole-induced phototoxicity masquerading as chronic graft-versus-host disease of the skin in allogeneic hematopoietic cell transplant recipients. Pseudoporphyria as a result of voriconazole use: a case report. Photoinduced lichenoid reaction by thioridazine. Phototoxic properties of neuroleptic drugs. Analysis of photosensitivity in Japanese cancer-bearing patients receiving photodynamic therapy with porfimer sodium (Photofrin). Moriwaki SI, Misawa J, Yoshinari Y, Yamada I, Takigawa M, Tokura Y. Duration of skin photosensitivity and incidence of photosensitivity reactions after administration of verteporfin. ![]() Photodynamic therapy and topical aminolevulinic acid: an overview. Cutaneous toxicities of new treatments for melanoma. 145(8):923-5.īoada A, Carrera C, Segura S, Collgros H, Pasquali P, Bodet D, et al. Cutaneous pigmentation after photosensitivity induced by vandetanib therapy. Statin-induced Ro/SSa-positive subacute cutaneous lupus erythematosus. Celecoxib-induced photoallergic drug eruption. Yazici AC, Baz K, Ikizoglu G, Kokturk A, Uzumlu H, Tataroglu C. Naproxen-induced lichen planus: report of 55 cases. Günes AT, Fetil E, Ilknur T, Birgin B, Ozkan S. Ketoprofen-induced photoallergic dermatitis. Drug-induced phototoxicity an early in vitro identification of phototoxic potential of new drug entities in drug discovery and development. Extensive lichenoid drug eruption due to glyburide: a case report and review of the literature. Photosensitivity and cancer immune-targeted therapies. Lembo S, Raimondo A, Conti V, Venturini M. Analysis of dermatologic events in vemurafenib-treated patients with melanoma. Lacouture ME, Duvic M, Hauschild A, et al. Prospective study of cutaneous side-effects associated with the BRAF inhibitor vemurafenib: a study of 42 patients. ![]() Vemurafenib: an unusual UVA-induced photosensitivity. Comparison of racial distribution of photodermatoses in USA academic dermatology clinics: A multicenter retrospective analysis of 1080 patients over a 10-year period. Hamel R, Mohammad TF, Chahine A, Joselow A, Vick G, Radosta S, et al. Updated Evaluation of the Safety, Efficacy and Tolerability of Pirfenidone in the Treatment of Idiopathic Pulmonary Fibrosis. 2020 Jan.Įberlein B, Biedermann T, Hein R, Posch C. Shedding light on drug photosensitivity reactions. Patients who show no signs of improvement may require the use of immunosuppressive agents (eg, azathioprine, cyclosporine). Paradoxically, psoralen UV-A (PUVA) and narrow band UV-B have been used, although relapse is common. Emollients, topical steroids, systemic steroids, and (at times) hydroxychloroquine. The treatment of persistent light reactivity involves the avoidance of contact with exacerbating agents and photoallergens. Although systemic drugs (eg, thiazides, quinidine) have been implicated as causes of persistent light reactivity, sunscreens, halogenated salicylanilides and musk ambrette are the most frequent causes. Some patients confine themselves to darkened rooms because of their severe photosensitivity. The photosensitivity can be incapacitating because the patients are sensitive to light not only in the UV-A range but also in both the UV-B and visible ranges. Initially, persistent light reactivity is misdiagnosed as atopic dermatitis or a lichenoid drug reaction. The disease may involve all sun-exposed areas and spread to covered areas of skin. In patients with persistent light reactivity, photosensitivity persists for months or years after the offending agent is eliminated. Persistent light reactivity is a form of chronic actinic dermatitis that occurs in patients with photoallergic contact dermatitis. ![]()
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